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Database.v7.1.5244.Workplace.Cracked-NoPE[XCRYPT] KeysOxidative stress and chronic obstructive pulmonary disease.
The aetiopathogenesis of COPD, the most frequent chronic obstructive pulmonary disease, is not yet fully elucidated. Chronic inflammation of the lung airways is one of the main pathophysiologic events that lead to COPD. Among other factors, cigarette smoke is responsible for this inflammation. The complex chemical composition of cigarette smoke leads to the production of superoxide anions which, through the enzymatic defence systems of the cells, are converted in to other species such as hydroxyl radicals. Oxidative stress and chronic inflammation play a key role in the pathophysiology of COPD. In this paper, we review the physiopathology of COPD, focusing on the changes in the antioxidant system of lung tissue.[The diagnostic value of pleural biopsy for pleural effusion associated with collagen disease].
Pleural biopsy was performed on 20 patients with pleural effusion associated with collagen disease. The purpose of the present study is to evaluate the diagnostic value of pleural biopsy. Mean pleural thickness was 1.5 mm and no definite thickening of pleura was observed. The pathologic findings of the biopsy specimen was classified into four categories: subepithelial fibrosis (SELF), pleuritis (PL), organizing pneumonia (OP) and normal. The SELF and PL was found in 16 (80%) and 9 (45%), respectively. Pathologic diagnosis was most frequently obtained when PL was observed. But PL was not observed in all the cases and only 3 cases were diagnosed as OP. OP was found in 7 (35%) out of 20 cases with collagen disease. Although biopsy demonstrated atypical findings such as OP, the specific diagnosis by pleural biopsy was not obtained in all cases with collagen disease. This indicated that pleural biopsy is a useful method for differential diagnosis of collagen disease associated with pleural effusion.#pragma once

#include “envoy/config/core/v3/grpc_service.pb.h”
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